Nursing Chapter 6 3 min read

Cardiovascular Nursing: Heart Failure, MI, and Cardiac Monitoring

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Oiyo Contributor
6/13

Chapter 6: Cardiovascular Nursing — Heart Failure, MI, and Cardiac Monitoring

Cardiovascular disease is the leading cause of death worldwide. Nurses in every setting — from ICU to home health — encounter patients with heart failure, coronary artery disease, and arrhythmias. Rapid, accurate assessment and targeted interventions are life-saving.

Heart Failure: Left vs. Right

Heart failure (HF) occurs when the heart cannot pump adequate blood to meet the body’s metabolic needs. Understanding which ventricle is failing guides assessment and interventions.

FeatureLeft-Sided HFRight-Sided HF
MechanismLV fails → blood backs into pulmonary circulationRV fails → blood backs into systemic venous circulation
Hallmark symptomDyspnea, orthopnea, PNDPeripheral edema, JVD, ascites
Breath soundsCrackles (pulmonary edema)Clear (unless biventricular)
WeightDaily weight gain (>2 lbs/day = notify MD)Same
Key assessmentSpO₂, respiratory rateDaily weight, peripheral edema, JVD

NYHA Classification: Class I (no symptoms with activity) → Class IV (symptoms at rest).

Myocardial Infarction (MI) Nursing Care

MI occurs when coronary artery occlusion causes myocardial ischemia leading to cell death. Nursing priorities follow MONA (now evidence-updated):

  • Morphine — use cautiously (associated with worse outcomes in NSTEMI)
  • Oxygen — only if SpO₂ < 90%
  • Nitroglycerin — for chest pain relief (hold if SBP < 90, recent PDE-5 inhibitor use)
  • Aspirin — 325 mg chewable immediately unless contraindicated

Key nursing actions: 12-lead ECG within 10 minutes of symptom onset, IV access, continuous cardiac monitoring, labs (troponin, CK-MB, BNP), prepare for PCI or thrombolytics.

STEMI vs. NSTEMI: STEMI shows ST elevation; requires emergent PCI (door-to-balloon ≤90 minutes). NSTEMI has elevated troponin without ST elevation.

ECG Basics

ComponentNormal ValueClinical Significance
P wave< 0.12 secAtrial depolarization
PR interval0.12–0.20 secAV conduction time
QRS complex< 0.12 secVentricular depolarization
QT interval< 0.44 sec (corrected)Ventricular repolarization; prolonged QTc = TdP risk
ST segmentIsoelectricST elevation = STEMI; ST depression = ischemia

Cardiac Medications

Digoxin (cardiac glycoside): increases contractility, slows AV conduction. Check apical pulse for 1 full minute before administering — hold if < 60 bpm in adults. Signs of toxicity: bradycardia, N/V, visual disturbances (yellow-green halos), arrhythmias. Therapeutic level: 0.5–2.0 ng/mL. Hypokalemia increases toxicity risk.

Beta-blockers (metoprolol, carvedilol): reduce heart rate and myocardial oxygen demand. Hold if HR < 50 or SBP < 90. Do not abruptly discontinue.

ACE Inhibitors/ARBs: reduce afterload and prevent cardiac remodeling in HF. Monitor for hypotension, hyperkalemia, renal function. ACE inhibitors cause dry cough (switch to ARB).

Key Checklist

  • Compare left-sided and right-sided heart failure: mechanisms, hallmark symptoms, and priority nursing assessments
  • Describe the nursing actions to take within the first 10 minutes of a suspected MI (MONA + diagnostics)
  • State the apical pulse threshold for holding digoxin and list three signs of digoxin toxicity
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