Cardiovascular Nursing: Heart Failure, MI, and Cardiac Monitoring
Chapter 6: Cardiovascular Nursing — Heart Failure, MI, and Cardiac Monitoring
Cardiovascular disease is the leading cause of death worldwide. Nurses in every setting — from ICU to home health — encounter patients with heart failure, coronary artery disease, and arrhythmias. Rapid, accurate assessment and targeted interventions are life-saving.
Heart Failure: Left vs. Right
Heart failure (HF) occurs when the heart cannot pump adequate blood to meet the body’s metabolic needs. Understanding which ventricle is failing guides assessment and interventions.
| Feature | Left-Sided HF | Right-Sided HF |
|---|---|---|
| Mechanism | LV fails → blood backs into pulmonary circulation | RV fails → blood backs into systemic venous circulation |
| Hallmark symptom | Dyspnea, orthopnea, PND | Peripheral edema, JVD, ascites |
| Breath sounds | Crackles (pulmonary edema) | Clear (unless biventricular) |
| Weight | Daily weight gain (>2 lbs/day = notify MD) | Same |
| Key assessment | SpO₂, respiratory rate | Daily weight, peripheral edema, JVD |
NYHA Classification: Class I (no symptoms with activity) → Class IV (symptoms at rest).
Myocardial Infarction (MI) Nursing Care
MI occurs when coronary artery occlusion causes myocardial ischemia leading to cell death. Nursing priorities follow MONA (now evidence-updated):
- Morphine — use cautiously (associated with worse outcomes in NSTEMI)
- Oxygen — only if SpO₂ < 90%
- Nitroglycerin — for chest pain relief (hold if SBP < 90, recent PDE-5 inhibitor use)
- Aspirin — 325 mg chewable immediately unless contraindicated
Key nursing actions: 12-lead ECG within 10 minutes of symptom onset, IV access, continuous cardiac monitoring, labs (troponin, CK-MB, BNP), prepare for PCI or thrombolytics.
STEMI vs. NSTEMI: STEMI shows ST elevation; requires emergent PCI (door-to-balloon ≤90 minutes). NSTEMI has elevated troponin without ST elevation.
ECG Basics
| Component | Normal Value | Clinical Significance |
|---|---|---|
| P wave | < 0.12 sec | Atrial depolarization |
| PR interval | 0.12–0.20 sec | AV conduction time |
| QRS complex | < 0.12 sec | Ventricular depolarization |
| QT interval | < 0.44 sec (corrected) | Ventricular repolarization; prolonged QTc = TdP risk |
| ST segment | Isoelectric | ST elevation = STEMI; ST depression = ischemia |
Cardiac Medications
Digoxin (cardiac glycoside): increases contractility, slows AV conduction. Check apical pulse for 1 full minute before administering — hold if < 60 bpm in adults. Signs of toxicity: bradycardia, N/V, visual disturbances (yellow-green halos), arrhythmias. Therapeutic level: 0.5–2.0 ng/mL. Hypokalemia increases toxicity risk.
Beta-blockers (metoprolol, carvedilol): reduce heart rate and myocardial oxygen demand. Hold if HR < 50 or SBP < 90. Do not abruptly discontinue.
ACE Inhibitors/ARBs: reduce afterload and prevent cardiac remodeling in HF. Monitor for hypotension, hyperkalemia, renal function. ACE inhibitors cause dry cough (switch to ARB).
Key Checklist
- Compare left-sided and right-sided heart failure: mechanisms, hallmark symptoms, and priority nursing assessments
- Describe the nursing actions to take within the first 10 minutes of a suspected MI (MONA + diagnostics)
- State the apical pulse threshold for holding digoxin and list three signs of digoxin toxicity
Oiyo
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